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My first question is I want to see images of Nutrisystem diet food? Thanks in advance for any response. Another question I got... I used to have this permanent red rash on my left buttock, for last 10 years or so. Even had a gf who was a bit weirded out by it, and wanted me to figure out what it was (but I never bothered...and docs never seemed bothered by it during exams although I forgot to ask them about it). is now almost completely gone. Any idea on what it was and if weight loss made it go away?..

Comments (12)

That's a good question. I'm not sure what is the right answer. I'll do some poking around and get back to you if I got an anything. You should email the people at Nutrisystem as they probably could answer your Nutrisystem question..

Comment #1

I surely don't know, but it seems that under calorie deprivation, your whole body seems to function better. All the systems get a tune-up. How's that for scientific?.


Comment #2

Uhhhh... I don't think I'd care to speculate about your butt cheeks..


Comment #3

It's not calorie deprivation, it's eating better quality food. More veggies, less junk. I would say that has a LOT to do with it..


Comment #4

I had some discolored skin on my shins that cleared up as well. I had it ever since I played Golf in Scotland and went to look for a wayward ball in the heather....I think Gordo is right. Your immune system got a tune up and cured the problem. Enjoy the new you...

Comment #5

Improved circulation? Or maybe you aren't sitting on your *** as much?..

Comment #6

Maybe you keep your wallet in your left pocket and when you were AcubictonDp, the wallet made an imprint?..

Comment #7


Better? I dunno I thought it was funny...but i'm slightly insane..and so am i..

Comment #8

It was good. Now hang in there and stay 100%. No binge and starve. Big salads.....

Comment #9

There is absolutely scientific evidence that suggests calorie deprivation, rather than any particular combination of nutrients, may be responsible Poly's <ahem> change..

It has been well established that long-term calorie deprivation (usually below 1,500 calories/day) seems to stimulate "survivor cells" in the body, which do all sorts of neat things, like fighting cancer and generally extending lifespan and slowing the normal aging process. If you google "low calorie longevity" you will find all sorts of information on it..

The science has been around since 1935 but is still developing. In fact, what scientists are doing nowadays is trying to devise a way to synthetically replicate what happens in the body when someone eats a calorie restricted diet, with the goal of being able to reap the health benefits without making an uncomfortable calorie restriction necessary (of course, those of us on Nutrisystem would tell you it's not uncomfortable to eat 1,200-1,500 calories a day, but for most people that's a huge leap)..

Proper nutrition is always a good thing, but if this is a condition that has existed for a long time, but cleared up after a long period of a low calorie diet, my guess would be that the process it stimulates helped attack and repair the condition..


Why A Low-Calorie Diet Extends Lifespans: Critical Enzyme Pair.


ScienceDaily (June 28, 2009) Experiment after experiment confirms that a diet on the brink of starvation expands lifespan in mice and many other species. But the molecular mechanism that links nutrition and survival is still poorly understood. Now, researchers at the Salk Institute for Biological Studies have identified a pivotal role for two enzymes that work together to determine the health benefits of diet restriction..

When lacking one enzyme or the other, roundworms kept on a severely calorie-restricted diet no longer live past their normal lifespan, they report in the June 24, 2009, advance online edition of the journal Nature..

"The only other known factor regulating longevity in response to diet restriction operates at the very end of the signaling cascade," said Howard Hughes Medical Investigator and senior author Andrew Dillin, Ph.D., an associate professor in the Molecular and Cell Biology Laboratory. "These two enzymes are further up the ladder, bringing us closer to the receptor that receives the signal for throwing the switch to promote a healthy lifespan.".

Identifying the receptor may allow researchers to design drugs that mimic the signal and could lead to new treatments for age-related diseases. This could enable us to reap the health benefits of calorie restriction without adhering to extreme diets in which the satisfying feel of a full stomach is strictly off limits..

Although lifestyle factors such as obesity clearly influence life expectancy, genetic factors are considered central to the process of aging. To date, there are only three known genetic networks that ensure youthfulness when manipulated. One centers on the insulin/insulin growth factor-1, which regulates metabolism and growth; the second is driven by mitochondria, the cell's power plants; and the third is linked to diet restriction..

But first author Andrea C. Carrano, Ph.D., a postdoctoral researcher in American Cancer Society Professor Tony Hunter's laboratory, hadn't set out to unravel the molecular connection between dietary restriction and increased lifespan when she started to investigate the role of the mammalian enzyme WWP-1. "I only knew that WWP-1 was a ubiquitin ligase and that mammalian cells contain three copies, which would make it difficult to study it's function.".

Ubiquitin ligases work in tandem with so called ubiquitin-conjugating enzymes to attach a chain of ubiquitin molecules to other proteins. This process, called ubiquitination, flags protein substrates for destruction but can also serve as a regulatory signal..

Since the laboratory roundworm Caenorhabditis elegans only contains one copy, Carrano teamed up with Salk researcher Dillin, who studies aging and longevity in C. elegans. Initial experiments revealed that worms without the WWP-1 gene seemed normal but were more susceptible to various forms of stress. "This finding was the first hint that WWP-1 might play a role in the aging process since mutations that affect stress very often correlate with longevity," she says..

Prompted by the findings, Carrano's next set of experiments focused on WWP-1's potential role in the regulation of lifespan. When she genetically engineered worms to overexpress WWP-1, well-fed worms lived on average 20 percent longer. Deleting PHA-4, which was discovered in Dillin's lab and so far is the only gene known to be essential for lifespan extension in response to diet restriction, abolished the life-extending effects of additional WWP-1 placing the ubiquitin ligase as a central rung on the same genetic ladder as PHA-4. Without WWP-1, cutting down on calories no longer staved off death..

When a study by others found that UBC-18 interacts with WWP-1, Carrano wondered whether it could play a role in diet-restriction-induced longevity as well. She first confirmed that the UBC-18 functions as an ubiquitin-conjugating enzyme and gives WWP-1 a hand. She then tested whether it played a role in lifespan regulation. "Overexpression of UBC-18 was not enough to extend the lifespan of worms but depleting it negated the effects of caloric restriction," says Carrano, who is busy looking for potential substrates of the UBC-18-WWP-1 ubiquitination complex..

"The WWP-1 pathway is highly conserved between worms and mammals and could play a role in the human aging process," says senior author Tony Hunter, Ph.D., a professor in the Molecular and Cell Biology Laboratory. "We didn't expect that this protein would be involved in the regulation of lifespan but it is very exciting when experiments lead you in a surprising direction.".

This work was supported by the National Institutes of Health, the Ellison Medical and Glenn Medical Foundations, the American Cancer Society and the Rossi Endowment..

Zheng Liu, Ph.D., a research associate, in the Dillin Laboratory also contributed to the work...

Comment #10

Now, THAT's what I'm talkin' about! Did I mention, I've got hair growing on the top of my head now?.


Comment #11

Well for years and years, I had a beard and folks would say to me: You're cultivating on your face, what I grow wild in my A$$..

Sure would like the hair on my head to start going again. Next step is a pony tail..

If I get down to 190, can we assume my hair will start to grow again?..

Comment #12

I am sure it's a combo of everything, calorie deprevation, increase water intake, exercise and better eating habits. I also know you have been swiming alot, maybe the clorine in the water is burning it off. Your whole system is just in better balance now. But what the heck do I know, I am just a CPA..

I know I had some brownish spots under my manboobs before I began and they are now gone..

My chest is getting hairier. I think I have the same number of hairs just smaller manboobs to cover!..

Comment #13

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This question was taken from a support group/message board and re-posted here so others can learn from it.